Title : Elucidating the role of aberrant macrophage activation in nephropathic cystinosis

نویسنده

  • Daryl Okamura
چکیده

OVERVIEW Overall, the project produced some insightful and intriguing results. The project was designed to further delineate the Ctns-/macrophage phenotype both in vitro (Aim 1) and confirming these phenotypic findings in vivo in the context of chronic kidney injury (Aim 2). The project progressed as expected for Aim 1 but we were confounded by the results from Aim 2. As discussed below, we found that despite in vitro studies of a hyperphagocytic, hyperinvasive macrophage phenotype that there was less fibrosis in Ctns ko chimeric mice during chronic kidney injury. We are still investigating the mechanism behind this discrepancy but hypothesize that it is due to an impaired ability to respond appropriately to chemotaxis during kidney injury. Studies are ongoing to investigate this hypothesis.

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تاریخ انتشار 2016